Resistant starch is a nondigestible fibre that ferments in the large intestine and has previously been found to boost metabolism in animal tests.
A 4-month randomised controlled experiment in persons with non-alcoholic fatty liver disease (NAFLD) found that regularly eating resistant starch
A novel method of optimising plant enzymes through bioengineering has expanded the understanding of how plant material can be transformed into biofuels, biochemicals, and other high-value goods.
Resistant starch is a nondigestible fibre that ferments in the large intestine, and it has previously been demonstrated in animal experiments to have a favourable effect on metabolism. A 4-month randomised controlled experiment in persons with non-alcoholic fatty liver disease (NAFLD) has fo
Bacteria utilise a number of strategies to combat the treatments meant to kill them. One of the most prevalent of these weapons is ribosome-modifying enzymes. These enzymes are becoming increasingly common, appearing in clinical samples from bacteria with varying drug resistances all over th
Bacteria use a variety of methods to fight off the medications designed to destroy them. Ribosome-modifying enzymes are one of these weapons that are most common and these enzymes are becoming more widespread
Cancer spreads in devious, almost military ways across the human body. It may, for example, alter our genetic make-up, take over certain cell-to-cell signalling mechanisms, and change essential enzymes to enhance tumour development, resistance to medicines, and expedite spread from the initi
According to new research from Ghent, Belgium, intravenous treatment with MK-3402, a metallo-beta-lactamase inhibitor, can be a successful technique in combating antimicrobial resistance. The findings will be presented at ASM Microbe 2023, the American Society for Microbiology's annual meeti
According to new research, neurodegeneration in brain cells may occur when the normal cellular cleansing mechanism failed owing to declining levels of a coenzyme related to niacin and deprives cells of energy.
A recent study from Cornell University offers a new perspective on how the chemotherapy drug etoposide delays and kills vital enzymes that support the growth of cancer cells.
New Cornell research is providing a fresh view into the ways a common chemotherapy agent, etoposide, stalls and poisons the essential enzymes that allow cancer cells to flourish.