New antiviral gene may stop virus clone

Worrying about getting older especially fearing future health problems may actually speed up aging at the cellular level, according to new research from NYU.

Human evolution has long been tied to growing brain size, and new research suggests prenatal hormones may have played a surprising role. By studying the relative lengths of the index and ring fingers, a marker of prenatal exposure to oestrogen and testosterone, researchers found that higher prenatal oestrogen exposure was associated with larger head size in newborn boys.

New research suggests that consistent aerobic exercise can help keep your brain biologically younger. Adults who exercised regularly for a year showed brains that appeared nearly a year younger than those who didn't change their habits.

A massive international brain study has revealed that memory decline with age isn't driven by a single brain region or gene, but by widespread structural changes across the brain that build up over time.

A high-fat diet does more than overload the liver with fat. New research from MIT shows that prolonged exposure to fatty foods can push liver cells into a survival mode that quietly raises the risk of cancer.

Alzheimer's has long been considered irreversible, but new research challenges that assumption. Scientists discovered that severe drops in the brain's energy supply help drive the disease, and restoring that balance can reverse damage, even in advanced cases.

A research team has found that specific immune cells can connect with muscle fibres in a lightning-fast, neuron-like way to promote healing.

Hypertension may impair the brain far earlier than previously understood -- even before a measurable rise in blood pressure occurs -- according to a new preclinical study from Weill Cornell Medicine. The changes help explain why hypertension is a major risk factor for developing cognitive disorders, such as vascular cognitive impairment and Alzheimer's disease.

Scientists discovered that tiny messengers released by fat tissue, called extracellular vesicles, can carry harmful signals that accelerate the buildup of amyloid-b plaques in the brain.

Researchers at the UCLA Health Jonsson Comprehensive Cancer Centre have identified a single protein, IGF2BP3, that links these two processes together in leukaemia cells. The protein alters how cells break down sugar, favouring a fast but inefficient energy pathway, while also modifying RNA that helps produce the proteins leukaemia cells need to survive and multiply.